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Although neutrophils are known to aggravate liver injury in a variety of diseases [115], an early study using antibodies against adhesion molecules to block neutrophil cytotoxicity did not protect suggesting that neutrophils do not aggravate APAP-induced liver injury [114]

These observations highlight the suitability of the HepG2 model to study these events and the necessity to define cellular sensors and signaling cascades involved in the NFT-induced increase of glutathione levels

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